Visual Psychophysics and Physiological Optics Nature of the Visual Loss in Observers With Leber’s Congenital Amaurosis Caused by Specific Mutations in RPE65

نویسندگان

  • Caterina Ripamonti
  • G. Bruce Henning
  • Robin R. Ali
  • James W. Bainbridge
  • Scott J. Robbie
  • Venki Sundaram
  • Vy A. Luong
  • L. Ingeborgh van den Born
  • Ingele Casteels
  • Thomy J. L. de Ravel
  • Anthony T. Moore
  • Andrew Stockman
چکیده

Citation: Ripamonti C, Henning GB, Ali RR, et al. Nature of the visual loss in observers with Leber’s congenital amaurosis caused by specific mutations in RPE65. Invest Ophthalmol Vis Sci. 2014;55:6817–6828. DOI: 10.1167/iovs.14-14923 PURPOSE. To characterize visual losses associated with genetic mutations in the RPE65 gene that cause defects in the RPE-specific isomerase, RPE65. RPE65 is an important component of the retinoid cycle that restores 11-cis-retinal after its photoisomerization to its all-trans form. The defects investigated here cause Leber’s congenital amaurosis (LCA2), an autosomal, recessively-inherited, severe, congenital-onset rod–cone dystrophy.

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Nature of the visual loss in observers with Leber's congenital amaurosis caused by specific mutations in RPE65.

PURPOSE To characterize visual losses associated with genetic mutations in the RPE65 gene that cause defects in the RPE-specific isomerase, RPE65. RPE65 is an important component of the retinoid cycle that restores 11-cis-retinal after its photoisomerization to its all-trans form. The defects investigated here cause Leber's congenital amaurosis (LCA2), an autosomal, recessively-inherited, sever...

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A Gene Scan Study of RPE65 in Chinese Patients with Leber Congenital Amaurosis

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W mutation in Rpe 65 leads to milder early - onset retinal dystrophy due to the generation of low levels of 11 - cis - retinal

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تاریخ انتشار 2014